missing translation for 'onlineSavingsMsg'
Learn More

GLRA1, Mouse, Clone: 3F1, Abnova™

Mouse monoclonal antibody raised against a partial recombinant GLRA1.

Brand:  Abnova H00002741-M05.100ug

Product Code. 16118734

  • € 349.00 / 100µg

Please to purchase this item. Need a web account? Register with us today!

This item is not returnable. View return policy

Description

Description

The protein encoded by this gene is a subunit of a pentameric inhibitory glycine receptor. The receptor mediates postsynaptic inhibition in the central nervous system. Defects in this gene are a cause of startle disease (STHE), also known as hereditary hyperekplexia or congenital stiff-person syndrome. Two transcript variants encoding different isoforms have been found for this gene

Sequence: IWKPDLFFANEKGAHFHEITTDNKLLRISRNGNVLYSIRITLTLACPMDLKNFPMDVQTCIMQLESFGYTMNDLIFEWQEQGAVQVADGLTLPQFILKEE
Specifications

Specifications

GLRA1
Monoclonal
Unconjugated
PBS with no preservative; pH 7.4
NM_000171
GLRA1
GLRA1 (NP_000162, 121 a.a. ∼ 220 a.a) partial recombinant protein with GST tag. MW of the GST tag alone is 26 KDa.
100 μg
Primary
Human
Antibody
IgG2a κ
ELISA, Western Blot
3F1
Mouse monoclonal antibody raised against a partial recombinant GLRA1.
GLRA1
MGC138878/MGC138879/STHE
Mouse
Affinity chromatography
RUO
2741
Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Certificates
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Thank You! Your feedback has been submitted. Fisher Scientific is always working to improve our content for you. We appreciate your feedback.