missing translation for 'onlineSavingsMsg'
Learn More
  1. Home
  2. Products
  3. Antibodies
  4. Primary Antibodies
  5. All Primary Antibodies
  6. 18796418

Mouse CD25/IL-2R alpha Alexa Fluor 594-conjugated Antibody, R&D Systems™
GREENER_CHOICE

Rat Monoclonal Antibody

Brand:  R&D Systems FAB9164T-025

Product Code. 18796418

  • € 187.00 / 25 tests

Please to purchase this item. Need a web account? Register with us today!

This item is not returnable. View return policy

Description

Description

CD25/IL-2R alpha Monoclonal specifically detects CD25/IL-2R alpha in Mouse samples. It is validated for Flow Cytometry.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

CD25/IL-2R alpha
Monoclonal
Alexa Fluor 594
Supplied in a saline solution containing BSA and Sodium Azide. with Sodium Azide
IL2RA
B6.1 mouse cytotoxic T cell line
25 Tests
Primary
Detects mouse CD25/IL-2 R alpha in flow cytometry.
Protect from light. Do not freeze. 12 months from date of receipt, 2 to 8 degreesC as supplied.
IgG1
Flow Cytometry
PC61.5.3
Flow Cytometry 5 uL/10^6 cells
CD25, CD25 antigen, IDDM10, IL-2 R alpha, IL-2 receptor subunit alpha, IL2R, IL2R alpha, IL-2R subunit alpha, IL-2Ra, IL2RA, IL-2-RA, IL2-RA, interleukin 2 receptor, alpha, interleukin-2 receptor subunit alpha, p55, TAC antigen, TCGFR
Rat
Protein A or G purified from hybridoma culture supernatant
RUO
3559
Mouse
Purified
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Certificates
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Thank You! Your feedback has been submitted. Fisher Scientific is always working to improve our content for you. We appreciate your feedback.